[HTML][HTML] Interleukin-12 and Th1 immune response in Crohn's disease: Pathogenetic relevance and therapeutic inplication
I Peluso, F Pallone, G Monteleone - World journal of …, 2006 - ncbi.nlm.nih.gov
Crohn's disease (CD) and ulcerative colitis (UC) are chronic inflammatory disorders of the
gastrointestinal tract that share clinical and pathological characteristics. The most accredited
hypothesis is that both CD and UC result from a deregulated mucosal immune response to
normal constituents of the gut microflora. Evidence, however, indicates that the main
pathological processes in these two diseases are distinct. In CD, the tissue-damaging
inflammatory reaction is driven by activated type 1 helper T-cell (Th1), whereas a humoral …
gastrointestinal tract that share clinical and pathological characteristics. The most accredited
hypothesis is that both CD and UC result from a deregulated mucosal immune response to
normal constituents of the gut microflora. Evidence, however, indicates that the main
pathological processes in these two diseases are distinct. In CD, the tissue-damaging
inflammatory reaction is driven by activated type 1 helper T-cell (Th1), whereas a humoral …
[HTML][HTML] Interleukin-12 and Th1 immune response in Crohn's disease: Pathogenetic relevance and therapeutic inplication
G Monteleone, F Pallone, I Peluso - World Journal of Gastroenterology, 2006 - wjgnet.com
Crohn's disease (CD) and ulcerative colitis (UC) are chronic inflammatory disorders of the
gastrointestinal tract that share clinical and pathological characteristics. The most accredited
hypothesis is that both CD and UC result from a deregulated mucosal immune response to
normal constituents of the gut microflora. Evidence, however, indicates that the main
pathological processes in these two diseases are distinct. In CD, the tissue-damaging
inflammatory reaction is driven by activated type 1 helper T-cell (Th1), whereas a humoral …
gastrointestinal tract that share clinical and pathological characteristics. The most accredited
hypothesis is that both CD and UC result from a deregulated mucosal immune response to
normal constituents of the gut microflora. Evidence, however, indicates that the main
pathological processes in these two diseases are distinct. In CD, the tissue-damaging
inflammatory reaction is driven by activated type 1 helper T-cell (Th1), whereas a humoral …
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